We have observed that presentation-capable (i.e. MHC-II+) IMs were also the main producer of CXCL9 in the spleen (Fig 6D); these cells were directly infected and co-localised with CD4+ T cells in situ (Fig 6G), suggesting that IMs represent the convergent point between infection, antimicrobial control and T cell recruitment. This evidence concerns the gene CXCL9 and infection.