To further clarify the connection between ILKAP and ICAM-1, ILKAP and a catalytic inactive mutant of ILKAP (H154D) unable to inhibit ILK activity (Leung-Hagesteijn et al., 2001) were overexpressed in tumor cells and mICAM-1 and sICAM-1 levels were measured. Here, ILK is linked to neoplasm.