To investigate if the TUG homolog ASPL is expressed in human adipose tissue and determine its localization, isolated human adipose tissue from female patients with no diagnosed metabolic disorders (type 1 or type 2 diabetes, or thyroid gland dysfunction) were permeabilized and stained using primary ASPL‐specific and GLUT4‐specific antibodies, and the protein distribution was visualized by confocal laser‐scanning microscopy (CLSM; Fig. 1A, Fig. S2). Here, SLC2A4 is linked to metabolic disease.