For the nasal asthma module, G3BP stress granule assembly factor 1 (G3BP1), which promotes innate immune transcriptional responses via NF-kB and c-Jun N-terminal kinase pathways, [72] and the protein-coding gene InaD-like protein (INADL) involved in epithelial migration [73] were both identified as nasal key drivers that were also associated with asthma. This evidence concerns the gene G3BP1 and asthma.