In dopaminergic neurons from the midbrains of (alpha)‐synucleinopathy models, TFEB overexpression or post‐translational hyperactivation downstream of mTORC1 rescues the aggregation of (alpha)‐synuclein, halting the progression of neuronal loss (Decressac et al, 2013). This evidence concerns the gene TFEB and synucleinopathy.