When comparing groups with normal body weight, individuals with increased BMI and abdominal obesity (group 3), and a group with NAFLD (group 4), no additional changes in the risk factor profile were observed, except for the frequency of hyperinsulinemia and median insulin levels in NAFLD, which is consistent with the idea of the role of NAFLD in the regulation of carbohydrate metabolism [17]. This evidence concerns the gene INS and Abdominal obesity.