It has also been shown that STZ‐treated C57BL/6 mice following TauT gene knockout displayed more advanced diabetic nephropathy than STZ‐induced C57BL/6 mice, as evidenced by considerable GBM thickening, enlarged mesangial matrix, fibrous thickening of efferent and afferent arterioles with severe dilatation and arteriosclerosis, and major renal hypertrophy. Here, SLC6A6 is linked to diabetic kidney disease.