CLEC4A and influenza: Myeloid DCs, pDCs, andLangerhans cells isolated from human PBMCs showed similar CD8+ T cell priming ability when targeted with anti-DCIR-MART-1peptide or anti-DCIR-influenza matrix protein fusions.166 DCIR had a different intracellular routingthan other C-type lectins and did not preferentially colocalize witheither early, late, or recycling endosomes, nor with the ER or Golgi.167 In line with the poor association with theendolysosomal system, low CD4+ T cell responses were measuredupon ligand internalization.