The first indications that dysregulation of interphase CDK‐cyclin complexes might contribute to this aspect of tumor development came from observations of chromosomal translocations involving the CCND1 gene, which encodes cyclin D1, in a human parathyroid carcinoma and, most notably, B cell lymphomas (Hsi et al., 1996; Lesage et al., 2005; H. Liu, Wang, & Epner, 2004; Shane, 2001; Thomázy et al., 2002; Vasef et al., 1999; Zhao et al., 2014). The gene discussed is CCND1; the disease is neoplasm.