Next, to further investigate the tumor-suppressive function of N1DARP, we established LINC00261 and N1DARP overexpression models by stably transfecting lentiviruses containing full-length LINC00261 and N1DARP into Capan1, and constructed a Crispr/Cas9-mediated LINC00261 and N1DARP knockout model by deleting full-length LINC00261 and partial exon 4 of LINC00261 containing full-length N1DARP in Panc1 (Supplementary Fig. S3f). This evidence concerns the gene LINC00261 and neoplasm.