Our analysis revealed that B cells, dendritic cells (DCs), mast cells, neutrophils, T helper cells, Th2 cells, tumor-infiltrating lymphocytes (TILs), antigen-presenting cell (APC) co-inhibition, and type II interferon (IFN) response exhibit varying involvement in the invasion of low- and high-risk gastric cancer subtypes (Figures 8D, E). Here, IFNA1 is linked to neoplasm.