Finally, in order to understand the mechanisms associated with the increase of 2-AA-LPC in the plaques of diabetic patients, we examined the association, through a correlation matrix, between 2AA-LPC, the biomarkers measured within the plaque (LPC and oxysterols) with plasma markers related to hyperglycemia (HbA1c), dyslipidemia (triglycerides), inflammation (CRP), and oxidative stress (7-β-OH-cholesterol). The gene discussed is CRP; the disease is Hyperglycemia.