NRP1 and neoplasm: The selected targets were patient-specific, as the analysis of another individual’s HPV+ OPSCC, showed that although some interactions, such as VEGFA/NRP1 and VEGFA/GPC1 were present, the most active L-Rs in the tumor were VEGFA/NRP2 and EGFR-related pathways (Fig. 3i, Supplementary Figure 17).