EP300 and ovarian carcinoma: Importantly, CRISPR‐interference and CRISPR‐deletion systems were used to functionally screen 86 SEs and identify new SEs with oncogenic functions.[10] Encouraged by these outstanding results, we further conducted H3K4me1, H3K4me3, H3K27Ac, polII, and EP300 ChIP‐seq experiments in six ovarian cancer cell lines and normal ovarian epithelial cells.