Importantly, CRISPR‐interference and CRISPR‐deletion systems were used to functionally screen 86 SEs and identify new SEs with oncogenic functions.[10] Encouraged by these outstanding results, we further conducted H3K4me1, H3K4me3, H3K27Ac, polII, and EP300 ChIP‐seq experiments in six ovarian cancer cell lines and normal ovarian epithelial cells. This evidence concerns the gene EP300 and ovarian cancer.