Shang Hongxia et al. (Shang et al., 2014) proved that HPS could reduce the serum levels of low-density lipoprotein cholesterol (LDL-C), aspartate aminotransferase (AST), triacylglycerol, and alanine aminotransferase (ALT) in NAFLD rats but increases the levels of high-density lipoprotein cholesterol (HDL-C) and the expression of SOD-1 gene in the liver tissue, as well as regulates the lipid metabolism disorder in NAFLD rats. The gene discussed is GPT; the disease is metabolic dysfunction-associated steatotic liver disease.