One of the chief causes includes genetic polymorphisms in anion transporters such as URAT1/SLC22A12, GLUT9/SLC2A9, and ABCG2/BCRP [2,7], among which defect in ABCG2 exporter is the prime cause of hyperuricemia and gout by decreasing renal urate excretion [2]. Lifestyle and diet preferences such as a diet rich in fats, meats, seafood, fructose, and alcohol can be important causes of the explosion in hyperuricemia incidence. Here, SLC22A12 is linked to gout.