Similarly, in non‐small cell lung cancer, the activation of PPARα ligand reduced the production of proangiogenic epoxyeicosatrienoic acids (EET) and increased the hydroxyl 11‐hydroxyeicosatetraenoic acids (11‐HETE) through Cyp2c44‐EETs system, thus inhibiting tumour progression and metastasis (Figure 2A).38 This evidence concerns the gene PPARA and neoplasm.