It also increased the number and toxicity of tumor-infiltrating T cells and enhanced antitumor immune responses.243 Moreover, inhibition of m6A-specific binding protein YTHDF1 can enhance tumor immunity like MRD.243 In EBV (Epstein-Barr virus)-infected tumors such as Burkitt’s lymphoma, Met metabolism helps shape B-cell immortalization required to regulate EBV-latent genes. Here, YTHDF1 is linked to neoplasm.