Also, the symptoms of myasthenia could be alleviated by drugs on myosin immunized experimental autoimmune myositis model, like butylphthalide, while the mechanism of butylphthalide is based on the increase of Ca2+-ATPase activities of muscle mitochondrial membrane and the decrease of IFN-γ (Interferon-γ) mRNA expression (Chen et al., 2018). This evidence concerns the gene IFNG and Fatigable weakness.