To further identify RTKs active in ALK-driven NB cells, we employed a human phospho-RTK array (layout in Supplementary Figure S1A), observing that both InsR and IGF1R were active in CLB-BAR and NB1 cells, and the signal intensity of pInsR was weaker than that of pIGF1R, particularly in NB1 cells (Figure 1A). This evidence concerns the gene IGF1R and neuroblastoma.