The examination of mRNA for LPL revealed its greatest abundance in the NCAD(+) group (+132%, NCAD(+) vs. NCAD(−), p < 0.05; NCAD(+) vs. CAD(−) +74%, p < 0.05; NCAD(+) vs. CAD(+) +164%, p < 0.05, Figure 5B). This evidence concerns the gene LPL and coronary artery disorder.