These species and their metabolites interact with other biomolecules and enhance the accumulation of lipid peroxides, such as MDA and HNE, disrupt membrane permeability and ion homeostasis, promote the formation of several cytokines, including IL-8, IL-6, and I -1β, in epithelial and monocyte cells [30,62], and thus contribute to the inflammatory storm observed in COVID-19 patients. Here, CXCL8 is linked to COVID-19.