APOE and coronary artery disorder: Currently, research on CAD genetics and genomics is burgeoning and tremendous success has been achieved in elucidating potential causal genetic loci and responsible mutations (single nucleotide polymorphisms, SNPs) that can significantly modulate the risk for CAD occurrence, including the genes responsible for the expression of the atherogenic lipoprotein(a), apolipoprotein E or for the defects in cholesterol metabolism which might even have a causal role for CAD [2,4,5,6,7].