The homotypic (PKD/PKD and CU062/CU062) and heterotypic (PKD/CU062) interactions were not lectin-like; they were specific to the PKD repeats in PC1 and not the repeats in the dyslexia-associated protein, the KIAA0319-like protein (KIAA0319L); see Figures S4 and S5. This evidence concerns the gene KIAA0319L and dyslexia.