GATA2 and hemophagocytic syndrome: Current reports of HLH caused by GATA2 deficiency remain scarce, and we would venture to hypothesize that most variants of these HLH patients affected zinc finger domain function or resulted in loss of the zinc finger domain due to premature termination of translation, and the HLH onset was induced by the infections of bacteria or virus (7, 11–17) (Table 1).