These results are in agreement with those obtained in a similar BD experimental model by Ojeda et al. [48], where BD exposure led to OS, inflammation, and apoptosis in cardiac myocytes and to an increase in the endothelial markers CTGF, VEGF, tPAI-1, and cav-1, joint to a higher blood pressure and heart rate. The gene discussed is CCN2; the disease is Behcet disease.