CD69 and neoplasm: There was also an increase in the percentage of killer‐cell lectin‐like receptor G1 (KLRG1)+ cells observed within the CD44dim CD69− CD8+ T‐cell compartment (Fig 3E and F), characteristic of terminally exhausted T cells, most likely resulting from chronic TCR stimulation and/or checkpoint protein signaling, at this dynamic stage of T‐cell mediated tumor regression.