showed that METTL3 overexpression alleviated NASH by suppressing the expression of CD36 and C-C motif chemokine ligand 2 (CCL2) (75); however, METTL3 regulated CD36 and CCL2 expression at the transcriptional level, rather than through m6A modification. This evidence concerns the gene CD36 and metabolic dysfunction-associated steatohepatitis.