Patients with functional AKI without evidence of tubular damage (NGAL-/SCr +) were more likely to have transient AKI (i.e., resolved by Day 3), while those with evidence of tubular damage (i.e. NGAL +) were more likely to have AKI on Day 3, regardless of functional biomarker status (i.e., SCr + or SCr-) [57]. The gene discussed is LCN2; the disease is acute kidney injury.