We found that in the prostate tumor the fusion consequently resulted in increased expression of a truncated FOXP2 protein that retained the complete FOXP2 functional domains (Spiteri et al., 2007; Hannenhalli and Kaestner, 2009; Lai et al., 2001; Shu et al., 2001), but had an aberrant C-terminus (Figure 1C and D, Figure 1—figure supplement 1F and G). Here, FOXP2 is linked to prostate neoplasm.