SLC2A1 and acute kidney injury: Although renal tubular epithelial cells (RTECs) derive little energy from glycolysis under physiological conditions, it was discovered that during the initial phase of acute kidney injury (AKI), upregulated hypoxia‐inducible factors,37, 38 among others, can activate the glycolytic pathway in RTECs by upregulating key glycolytic pathway enzymes such as membrane glucose transporter protein‐1 (Glut‐1)39 and pyruvate kinase M2 (M2‐PK).40