Nonneural circulating CRTAC1 presumably has the same activity as truncated neural CRTAC1 but to connect low CRTAC1 levels to CNS symptoms in COVID‐19 such as anosmia, it would need to be shown that the nonneural form crosses the blood–brain barrier and contributes functionally to the pool of CRTAC1 regulating neuronal connections. This evidence concerns the gene CRTAC1 and COVID-19.