Recent data suggested that some of the genes mutated in dyskeratosis congenita or Fanconi anemia may affect ribosomal function (Benyelles et al., 2019; Gueiderikh et al., 2021) and frameshift TP53 mutations cause a pure red cell aplasia resembling Diamond–Blackfan anemia, together with relatively short telomeres (Toki et al., 2018). Here, TP53 is linked to dyskeratosis congenita.