For example, pathogenesis-related PR genes are induced early, at high levels, during incompatible host-pathogen interactions than during compatible interactions.23 Pathogenesis-related proteins do not usually accumulate in healthy plants but are induced by pathogen infection or related stresses and improve the defensive capacity of plants.24 In this study, the expression levels of PR protein 3 (PR3) were determined in infected plants at 11, 14, and 21 dpi. This evidence concerns the gene PRTN3 and infection.