For huCLL samples, we were able to collect enough amount of CD14+ cells (attributable to unstimulated monocyte/macrophage system or precursor of NLCs—pre‐NLCs) ready to use as effector cells in co‐culture analysis and redirect these unstimulated autologous effector cells against huCLL cancer cells, since Siglec‐10 levels seemed to be higher if compared to donor‐derived counterparts (Figure S9C). This evidence concerns the gene SIGLEC10 and cancer.