CXCL9/10, the ligands for CXCR3 and induced by IFNγ, were overexpressed by monocytes in patients with CIP versus patients without CIP, which was also enriched in patients with colitis induced by ICIs [30], are important for recruiting effector T cells to tumors, which would be expected to have positive effects on anti-tumor immunity [46]. Here, CXCR3 is linked to hereditary sensory and autonomic neuropathy.