CXCL9 and hereditary sensory and autonomic neuropathy: Meantime, CXCL9+monocytes and LAMP3+DCs were also elevated in CIP, and pseduotime trajectories showed that LAMP3+DCs were derived from the differentiation of hyperinflammatory CXCL9+monocytes, whereas the differentiation routine of anti-inflammatory alveolar macrophages was blocked, indicating a hyperinflammatory environment in CIP samples [25, 37].