Wild-type S. Typhimurium maintained higher fecal loads and elicited more pronounced enteropathy, while S.TmhilD loads declined during the 5 days of infection and caused attenuated enteropathy with delayed kinetics as judged by lipocalin-2 ELISA and by histopathology (Fig 2B–2D). This evidence concerns the gene LCN2 and Abnormal intestine morphology.