In contrast, recent studies have shown that neutrophil skin infections such as Staphylococcus aureus produce within 10 min of stimulation, and even when NETosis occurs, some neutrophils do not die but remain active, showing an immune response through crawling Toll-like receptor 2 (TLR2) and the protein of the compliment, a granular release network that extends the unstable pseudopod and extends it through the skin. Here, TLR2 is linked to skin infection.