Tominaga et al.20found that IL‐38 could increase the expression of anti‐fibrotic factor interferon‐γ, reduce the expression of tumor necrosis factor‐α (TNF‐α), and improve pulmonary fibrosis in Bleomycin (BLM) induced pulmonary fibrosis. The gene discussed is IL1F10; the disease is pulmonary fibrosis.