Anti‐CCL1 and anti‐CCL22 monoclonal neutralizing antibodies, but not anti‐CCL17, inhibited this chemotactic process significantly (Figure 8A,B), suggesting that the chemokines CCL1 and CCL22 may be key factors in the recruitment of CD4+ T cells from peripheral blood to infiltrate the diseased trachea in TS. The gene discussed is CCL17; the disease is Timothy syndrome.