To test the hypothesis that the DMPK 3’UTR CTGexp mutation leads to mis-splicing of ASD-risk genes, we analyzed human prefrontal cortex (Brodmann area 10; BA10) RNA-seq data generated from DM1 (unknown ASD status) and unaffected control samples (Supplementary Table 1)38. This evidence concerns the gene DMPK and myotonic dystrophy type 1.