All the associations reported in thisstudy were similarly reported for IgA and IgG in IBD, IgA nephropathy,and other autoimmune disease like RA, except for the sialylation ofthe O-glycans in RA and the bisection of IgG in CDvs HC (Figure 8).3,13–15,22,34,38,40,45,49 The changesin IgA glycosylation with IBD were mostly opposite of the associationsreported during pregnancy, similar to IgG where glycosylation changeswith inflammatory conditions were opposed to those occurring withpregnancy.13,14,34. This evidence concerns the gene CD79A and autoimmune disease.