EGFR and lung carcinoma: Comparison of proteome‐wide lysine methylation profiles reveals that the methylation level of DTPs increases on K116 of the PRC member Jarid2 in BC and lung cancer, and it is similar to that of EGFR inhibitor‐treated EGFR‐mutant NSCLC cells, thereby facilitating PRC2 stabilization and recruitment into chromatin.162