In patients with DYT-TOR1A dystonia, mutations in the TOR1A gene impairs the binding of TorsinA to LAP1/LULL1, thereby compromising the ability of TorsinA to hydrolyze ATP (Zhao et al., 2013; Sosa et al., 2014; Demircioglu et al., 2016). The gene discussed is TOR1A; the disease is Dystonia.