Currently, DYT- TOR1A dystonia is widely recognized as a neurodevelopmental disorder, and the relationship between TorsinA and mouse survival, as well as the human neurological phenotypes resulting from its mutation, suggests an important relationship between TorsinA and synaptic function and neurodevelopment (Dang et al., 2005; Goodchild et al., 2005; Kariminejad et al., 2017; Reichert et al., 2017; Isik et al., 2019). The gene discussed is TOR1A; the disease is neurodevelopmental disorder.