Studies on HDAC6’s function in the infection of viruses have mostly focused on three aspects: (a) controlling viral transport, fusion, and viral component release by modulating plasma membrane dynamics and the cytoskeleton (55, 56, 62, 63, 74, 77); (b) influencing host cells’ antiviral immune response (19, 23, 69); (c) tuning the breakdown of viral or host proteins in host cells through autophagy (64, 68, 78). Here, HDAC6 is linked to infection.