Conversely, HAS3 exhibited significant upregulation in CESC, cholangiocarcinoma (CHOL), esophageal carcinoma (ESCA), HNSC, KIRC, KIRP, LIHC, LUSC, pheochromocytoma and paraganglioma (PCPG), and UCEC tumors. This evidence concerns the gene HAS3 and carcinoma of esophagus.