SNHG7 and kidney disorder: In contrast, Br-MSCs + EXO, EXOs, and Br-MSCs’ transplantation caused a significant (p < 0.001) downstream regulation of mir-34a, mTOR, and AKT expression, and upstream regulation in SNHG-7, AMPK, and ULK-1 expression, respectively, compared with nephropathy and CM-treated groups (Figure 5A–F).