SLC12A1 and Hyperkalemia: An electrolyte disturbance was observed in a few patients; we have ignored hyperpotassemia (10 instances) on the grounds of possibly hemolyzed blood specimens because of the difficult venipunctures in some ASD kids, but the observed lowered sodium is more likely linked to a functional alteration in the NKCC2, a cotransporter protein of Na+, K+, and Cl− encoded via the SLC12A1 gene.