SNHG14 affects the Bcl-2 apoptosis pathway [145]; AFAP1-AS1 stimulates the overproduction of HER2, leading to treatment ineffectiveness [146]; AGAP2-AS1 is packaged in vesicles by hnRNPA2B1 [148] to link ELAVL1 in the receptor cell in order to modulate autophagy activity via the transcription of ATG10 [147]; and BCRT1 inhibits hsa-miR-1303 in order to induce the expression of PTBP3, a tumor promoter and target of hsa-miR-1303 [149]. Here, AFAP1 is linked to neoplasm.