When investigating the defense role of a pathogen-induced WAK gene from wheat chromosome 7D, designed as TaWAK7D, Qi et al. [41] suggested that such a gene positively participates in the defense against infection by the soilborne and necrotrophic fungus Rhizoctonia cerealis, through activating the expression of several pathogenesis-related (PR) genes, including Chitinase3, Chitinase4, PR1, PR17 and β-1,3-Glucanase. The gene discussed is TMEM37; the disease is infection.